The rodent prefrontal cortex maintains a “memory trace” for a gamma frequency (~ 40 Hz) rhythm
Location
D.P. Culp Center Ballroom
Start Date
4-5-2024 9:00 AM
End Date
4-5-2024 11:30 AM
Poster Number
135
Name of Project's Faculty Sponsor
Siva Digivalli
Faculty Sponsor's Department
Pharmaceutical Sciences
Competition Type
Competitive
Type
Poster Presentation
Presentation Category
Science, Technology and Engineering
Abstract or Artist's Statement
Working memory refers to one’s ability to temporarily keep and use a small amount of information for cognitive tasks. The prefrontal cortex is believed to be the primary region where such information is held temporarily. Working memory is known to be deficient in states of mental illness such as schizophrenia and is attributed to a reduction in NMDA-based neurotransmission. In our lab, we evaluate neural synchrony in response to rhythmic clicks as a marker for prefrontal cortical performance. In this study, we hypothesized that when two rhythmic inputs at 40 Hz are presented back-to-back with some time gap, the prefrontal cortex will continue to maintain a “memory trace” of the first train till it synchronizes to the second train. Evaluating progressively longer gaps, we show that a “memory trace” of the beat frequency, as recorded by the EEG, is maintained for at least 225 ms. Moreover, this memory trace was dose dependently disrupted by an NR2B selective NMDA antagonist, CP101,606 as well as the non-selective NMDA channel blocker, MK801. We conclude that rodent prefrontal cortex can maintain a “memory trace” of a rhythm up to several hundred milliseconds after stimulus and that this may involve the activation of the NR2B subtype of NMDA receptors.
The rodent prefrontal cortex maintains a “memory trace” for a gamma frequency (~ 40 Hz) rhythm
D.P. Culp Center Ballroom
Working memory refers to one’s ability to temporarily keep and use a small amount of information for cognitive tasks. The prefrontal cortex is believed to be the primary region where such information is held temporarily. Working memory is known to be deficient in states of mental illness such as schizophrenia and is attributed to a reduction in NMDA-based neurotransmission. In our lab, we evaluate neural synchrony in response to rhythmic clicks as a marker for prefrontal cortical performance. In this study, we hypothesized that when two rhythmic inputs at 40 Hz are presented back-to-back with some time gap, the prefrontal cortex will continue to maintain a “memory trace” of the first train till it synchronizes to the second train. Evaluating progressively longer gaps, we show that a “memory trace” of the beat frequency, as recorded by the EEG, is maintained for at least 225 ms. Moreover, this memory trace was dose dependently disrupted by an NR2B selective NMDA antagonist, CP101,606 as well as the non-selective NMDA channel blocker, MK801. We conclude that rodent prefrontal cortex can maintain a “memory trace” of a rhythm up to several hundred milliseconds after stimulus and that this may involve the activation of the NR2B subtype of NMDA receptors.